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tendon adhesion under the extensor retinaculum, secure
tendon-to-bone fixation, and appropriate rehabilitation at 6
weeks postoperation for tendon mobilization are the essential components.
An iatrogenic collapse of the medial longitudinal arch has
been a major concern in complete loss of TP tendon function, which may cause an increased calcaneal eversion and
decreased plantarflexion of the forefoot.17 Although adultacquired flatfoot deformity following TP tendon transfer has
been reported,12,18 these are limited to a few case reports.
The posterior tibial split tendon transfer to correct a varus
hindfoot deformity was reported to have the advantage in a
younger patient of minimizing the chance of a postoperative
flatfoot deformity.13 Mizel et al9 reported that no collapse of
the medial longitudinal arch and valgus hindfoot deformity
were found at an average follow-up of 74.9 months after TP
tendon transfer. Recently, Johnson et al6 reported there was
no indication that loss of the normal function of the tibialis
posterior muscle resulted in change in static stance foot
alignment 2 years after the Bridle procedure (modification
of the TP tendon transfer). Despite a concern regarding
adverse effects following complete sacrificing of the TP tendon, the present study also demonstrated no definitive radiographic nor clinical progression to postoperative flatfoot
deformity at intermediate-term follow-up. Because the
effects of TP tendon transfer on occurrence of an acquired
flatfoot or pes planovalgus deformity remain debatable,19-21
further long-term studies are needed to determine the risk of
loss of arch height after this procedure.
Omer11 reported that full active dorsiflexion is rarely
restored by TP tendon transfer alone. Because a transferred
muscle usually loses one grade of strength,1 full recovery of
dorsiflexion strength may be difficult despite a long period
of rehabilitation. Yeap et al20 reported that the torque generated by the transferred TP tendon for foot drop was about
30% compared to normal contralateral side, although 11 of
12 patients achieved clinically grade 4 or 5 muscle power of
dorsiflexion. Johnson et al6 also reported that patients had
only 18% of peak torque compared to the normal contralateral side after the Bridle procedure (tri-tendon anastomosis
of the peroneus longus, anterior and posterior tibial tendons). In the present study, a manual power grade of ankle
dorsiflexors significantly improved from a mean of 1.1 preoperatively to 3.9 at final follow-up. Mean peak torque (in
60 degrees/sec angular velocity) of dorsiflexors was 7.1 Nm
at final follow-up, which indicated that postoperative restoration of dorsiflexion strength was about 33% compared to
the control group, and 34.6% compared with the contralateral side. Despite incomplete dorsiflexion power, function
in daily activities and gait ability were satisfactorily
improved. All patients demonstrated the ability to dorsiflex
the foot during the swing phase, and only 1 patient (5.9%)
needed an ankle-foot orthosis for occupational activity.

Foot & Ankle International 38(6)
Whether the tendon transferred to another site can provide an active substitution for weak muscles or role of
tenodesis to dynamically fix a joint has always been a matter of concern.2,16,20 In terms of postoperative function, the
active substitution is known to be superior to a simple
tenodesis, because tenodesis would limit activities that
require a wide ROM and a fast switch from extension to
flexion of the involved joint.2 In a recent study regarding
the 3-dimensional gait analysis after TP tendon transfer to
correct footdrop, Dreher et al2 reported that TP tendon
transfer works as an active substitution, not as a tenodesis.
Although passive plantarflexion was not limited after surgery, active plantarflexion at push-off was significantly
reduced. Patients in the present study also showed a similar pattern of ROM limitation during the early postoperative period. However, we could identify the gradually
increasing active plantarflexion with improvement of calf
muscle weakness after muscle strengthening rehabilitation
and gait training.
The limitations of our study are as follows. First, physical examination including assessment of the ROM, manual
strength testing of all relevant musculotendinous units, and
sensory evaluation was performed only by the corresponding author. Therefore, we could not be completely convinced of the accuracy of physical examination, because the
reliability (verification of intraobserver reproducibility)
was not assessed. Second, this study included no objective
gait analysis. Dynamic EMG information during the entire
gait may have supported our conclusion that an out-ofphase tendon transfer became in-phase after surgical treatment and rehabilitation, as evidenced by active ankle
dorsiflexion during the swing phase of gait.

Conclusion
Anterior transfer of the tibialis posterior tendon appears to
be an effective surgical option for paralytic footdrop secondary to peroneal nerve palsy. Although restoration of dorsiflexion strength postoperatively was about 33% of a
normal ankle, function in daily activities and gait ability
were satisfactorily improved. In addition, tibialis posterior
tendon transfer demonstrated no definitive radiographic nor
clinical progression to postoperative flatfoot deformity at
intermediate-term follow-up.
Declaration of Conflicting Interests
The author(s) declared no potential conflicts of interest with respect
to the research, authorship, and/or publication of this article.

Funding
The author(s) received no financial support for the research,
authorship, and/or publication of this article.



Table of Contents for the Digital Edition of Foot & Ankle International - June 2017

Contents
Foot & Ankle International - June 2017 - Intro
Foot & Ankle International - June 2017 - Cover1
Foot & Ankle International - June 2017 - Cover2
Foot & Ankle International - June 2017 - i
Foot & Ankle International - June 2017 - ii
Foot & Ankle International - June 2017 - Contents
Foot & Ankle International - June 2017 - iv
Foot & Ankle International - June 2017 - v
Foot & Ankle International - June 2017 - vi
Foot & Ankle International - June 2017 - vii
Foot & Ankle International - June 2017 - viii
Foot & Ankle International - June 2017 - 1A
Foot & Ankle International - June 2017 - 1B
Foot & Ankle International - June 2017 - ix
Foot & Ankle International - June 2017 - x
Foot & Ankle International - June 2017 - xi
Foot & Ankle International - June 2017 - xii
Foot & Ankle International - June 2017 - 2A
Foot & Ankle International - June 2017 - 2B
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Foot & Ankle International - June 2017 - xv
Foot & Ankle International - June 2017 - xvi
Foot & Ankle International - June 2017 - 3A
Foot & Ankle International - June 2017 - 3B
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Foot & Ankle International - June 2017 - CT1
Foot & Ankle International - June 2017 - CT2
Foot & Ankle International - June 2017 - 4A
Foot & Ankle International - June 2017 - 4B
Foot & Ankle International - June 2017 - Cover3
Foot & Ankle International - June 2017 - Cover4
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https://www.nxtbook.com/nxtbooks/sage/hospitalpharmacy_february2018
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https://www.nxtbook.com/nxtbooks/sage/hospitalpharmacy_june2017
https://www.nxtbook.com/nxtbooks/sage/hospitalpharmacy_may2017
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https://www.nxtbook.com/nxtbooks/sage/fai_201607
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