ASH News Daily - Monday, December 12, 2011 - (Page A-1)
Celebrating 10 Years
ASH NEWS DAILY
53rd Annual Meeting of the American Society of Hematology
®
Issue 3, Section A
Monday, December 12, 2011
San Diego, CA
Read this issue online at
www.nxtbook.com/nxtbooks/ashnewsdaily2011_Monday/
Schedule
9:00 – 10:00 a.m.
E. Donnall Thomas Lecture
and Prize
San Diego Convention Center
Hall AB
10:30 a.m. – 12:00 noon
Education Spotlight Sessions
(ticketed sessions)
1:30 – 2:30 p.m.
Ernest Beutler Lecture and Prize
San Diego Convention Center
Hall AB
2:45 – 4:15 p.m.
Education Spotlight Sessions
(ticketed sessions)
6:00 – 8:00 p.m.
Poster Hall Reception
San Diego Convention Center
Hall GH
Looking ahead to Tuesday
7:00 a.m. – 1:30 p.m.
On-Site Registration
San Diego Convention Center
Sails Pavilion
7:30 a.m. – 9:00 a.m.
Simultaneous Sessions
7:30 a.m. – 9:00 a.m.
Late-Breaking Abstracts Session
San Diego Convention Center
Hall AB
Bone Marrow Harvest Time:
Are We Going Back to the OR?
By JoSeph Mikhael, Md,
Med
become a rare event in the
life of a hematologist. Indeed,
mobilizing stem cells
from sibling donors has
been the standard of care for
transplantation for over a
decade. The same approach,
however, for unrelated donors
is now in question after
Dr. Claudio Anasetti, H. Lee
Moffitt Cancer Center and
Research Institute, presented the first
abstract at the Plenary Scientific session
yesterday.
On behalf of the Blood and Mar-
H
row Transplant Clinical Trials Network
(BMT CTN), Dr. Anasetti revealed
the results of Protocol 0201,
a phase III, prospective, randomized
trial comparing bone marrow (BM)
transplants to filgrastim-mobilized
peripheral blood stem cells (PBSC).
MDS
Found It! One More Piece In The MDS Puzzle
By BarBara pro, Md
IN THIS SECTION
It Is All in the Genes
A-4
Find Cancer Early
– Really Early
A-6
Are We Finally Getting
Smarter When It Comes to
Stem Cell Transplant?
A-9
The Truth Behind “TRIM”
A-14
Spotlight on Lymphoma:
The Latest Biology
and Biologic Treatments
A-15
ing the heterogeneity of myelodysplastic
syndromes (MDS) and the
clonal evolution of these disorders.
However, like jumbled pieces from
a puzzle box, we have yet to sort
out the multitude of features that
will help us fully understand this
disease. The abstract presented
by Dr. Luca Malcovati, Policlinico
San Matteo and University of Pavia,
Italy, during yesterday’s Plenary
Scientific session reported the
identification of recurrent somatic
mutations in the SF3B1 gene in patients
with refractory anemia with
ring sideroblasts (RARS). This work
was presented on behalf of the
I
ncreasing evidence suggests
that chromosome abnormalities
play a critical role in determin-
International Cancer Genome Consortium
on Chronic Myeloid Disorders
Working Group and is the
result of a collaborative study with
participating centers in the United
Kingdom, Sweden, and Italy.
Dr. Malcovati began his presenta-
tion summarizing the data currently
available on somatic mutations associated
with MDS and their important
prognostic implications. RARS is a
specific subtype of MDS defined by
the presence of 15 percent or more
ring sideroblasts in the bone marrow.
Another (good) characteristic
of RARS is a very indolent clinical
course with a low risk of leukemic
evolution. Using parallel sequencing
technology, investigators tried
to identify somatically acquired
»» MDS Page A-6
Dr. Luca Malcovati presents during
Sunday’s Plenary Scientific session.
arvesting bone
marrow in the operating
room has
Dr. Claudio Anasetti presents his abstract during the Plenary Scientific session.
To the surprise of many in the field,
there was no survival difference between
the two groups at three years;
this is in stark contrast to previous
trials in sibling donors where PBSC
was superior to BM in decreasing relapse
and increasing survival.
As expected, however, PBSC does
increase the risk of chronic graft-versus-host
disease (GVHD). Although
rates of acute GVHD were similar,
chronic GVHD occurred in 53 percent
of patients with PBSC and in 40 percent
of those with BM (p=0.02). Why
such a difference? “I believe PBSC
cause more GVHD than marrow because
they contain about 20-50-fold
more T cells than marrow graft, and
we use the same approach for GVHD
prevention,” Dr. Anasetti commented.
»» BMT Page A-2
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